For decades, the “glucocentric” approach to Type 2 Diabetes has trapped us in a clinical paradox: to lower blood sugar, we force-feed glucose into resistant cells using insulin, often driving weight gain and worsening the very resistance we aim to treat. We are managing the number, but feeding the pathology.
But what if we could bypass the insulin receptor entirely?
In this week’s deep dive, The “Trojan Erythrocyte” Protocol, I propose a radical bioengineering breakthrough: transforming the humble Red Blood Cell from a passive carrier into an active “metabolic sink.” Imagine a system where genetically modified erythrocytes vacuum up excess plasma glucose and lock it away as inert, non-metabolizable polymers—turning the bloodstream itself into a glucose-disposal organ. No insulin. No weight gain. Just pure, mechanical sequestration.
Read on to explore how we might engineer the perfect metabolic trap.